M2I-1 disrupts the in vivo interaction between CDC20 and MAD2 and increases the sensitivities of cancer cell lines to anti-mitotic drugs via MCL-1s
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0049-5
HSP70 is required for the proper assembly of pericentriolar material and function of mitotic centrosomes
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0047-7
p27kip1 at the crossroad between actin and microtubule dynamics
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0045-9
MicroRNAs’ control of cancer cell dormancy
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0054-8
FGF-induced LHX9 regulates the progression and metastasis of osteosarcoma via FRS2/TGF-β/β-catenin pathway
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0056-6
A novel resveratrol derivative induces mitotic arrest, centrosome fragmentation and cancer cell death by inhibiting γ-tubulin
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0046-8
Septin and Ras regulate cytokinetic abscission in detached cells
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0051-y
Nucleoporin Nup58 localizes to centrosomes and mid-bodies during mitosis
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0050-z
Overexpression of P16 reversed the MDR1-mediated DDP resistance in the cervical adenocarcinoma by activating the ERK1/2 signaling pathway
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0048-6
Mechanisms for the temporal regulation of substrate ubiquitination by the anaphase-promoting complex/cyclosome
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0057-5
The essentiality landscape of cell cycle related genes in human pluripotent and cancer cells
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0058-4
CRY arrests Cop1 to regulate circadian rhythms in mammals
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0055-7
Phosphatidylinositol-5-phosphate 4-kinase gamma accumulates at the spindle pole and prevents microtubule depolymerization
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0053-9
Correction to: Reactivation of TAp73 tumor suppressor by protoporphyrin IX, a metabolite of aminolevulinic acid, induces apoptosis in TP53-deficient cancer cells
来源期刊:Cell DivisionDOI:10.1186/s13008-019-0052-x