ACTA NEUROPATHOLOGICA
ISSN:0001-6322

ACTA NEUROPATHOLOGICA

ACTA NEUROPATHOL
学科领域:医学
是否预警:不在预警名单内
是否OA:
录用周期:偏慢,4-8周
新锐分区:医学1区
年发文量:162
影响因子:9.3
JCR分区:Q1

基本信息

Acta Neuropathologica旨在发表有关神经疾病病理学的最佳论文,以及使用体外和体内模型进行的分子和细胞机制的实验研究,通过对人体组织的分析得到理想的验证。该杂志发表原始论文,评论文章,病例报告,和科学通信 (信件)。提交出版的手稿必须包含一份声明,大意是所有人类研究都经过了适当的伦理委员会的审查,因此是根据《赫尔辛基1964宣言》适当版本中规定的伦理标准进行的。案文中还应明确指出,所有人在纳入研究之前均已知情同意。可能披露被研究对象身份的细节应省略。动物实验报告必须说明遵循 “实验动物护理原则” (美国国立卫生研究院第86-23号出版物,1985修订),以及适用的具体国家法律 (例如德国保护动物法律的当前版本)。编辑保留拒绝不符合上述要求的手稿的权利。提交人将对虚假陈述或未能满足上述要求负责。
0001-6322SCIE/Scopus收录
9.3
9.9
2026年3月发布
点击查看历史分区趋势    >
大类学科小类学科Top期刊综述期刊
医学1区
CLINICAL NEUROLOGY 临床神经病学
1区
NEUROSCIENCES 神经科学
1区
PATHOLOGY 病理学
1区
N/A
WOS期刊SCI分区  2024-2025最新升级版
按JIF指标学科分区收集子录JIF分区JIF排名百分位
学科:CLINICAL NEUROLOGY
SCIE
Q1
8/286
学科:NEUROSCIENCES
SCIE
Q1
16/314
学科:PATHOLOGY
SCIE
Q1
3/90
按JCR指标学科分区收集子录JCR分区JCR排名百分位
学科:CLINICAL NEUROLOGY
SCIE
Q1
7/286
学科:NEUROSCIENCES
SCIE
Q1
9/314
学科:PATHOLOGY
SCIE
Q1
3/90
146
162
2%较难偏慢,4-8周-医学-病理学
6.5%
时间预警情况
2026年03月发布的新锐学术版不在预警名单中
2025年03月发布的2025版不在预警名单中
2024年02月发布的2024版不在预警名单中
2023年01月发布的2023版不在预警名单中
2021年12月发布的2021版不在预警名单中
2020年12月发布的2020版不在预警名单中
93.21%74.79%10.4%
CiteScore:17.90
SJR:4.483
SNIP:2.244
学科类别分区排名百分位
大类:Neuroscience
小类:Cellular and Molecular Neuroscience
Q1
5 / 100
大类:Neuroscience
小类:Neurology (clinical)
Q1
9 / 400
大类:Neuroscience
小类:Pathology and Forensic Medicine
Q1
2 / 204

期刊高被引文献

Second-generation molecular subgrouping of medulloblastoma: an international meta-analysis of Group 3 and Group 4 subtypes
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02020-0
A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02026-8
FUS pathology in ALS is linked to alterations in multiple ALS-associated proteins and rescued by drugs stimulating autophagy
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01998-x
H3.3 K27M depletion increases differentiation and extends latency of diffuse intrinsic pontine glioma growth in vivo
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01975-4
LRRK2 modifies α-syn pathology and spread in mouse models and human neurons
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01995-0
Selective vulnerability in α-synucleinopathies
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02010-2
SORL1 genetic variants and Alzheimer disease risk: a literature review and meta-analysis of sequencing data
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01991-4
Precise detection of low-level somatic mutation in resected epilepsy brain tissue
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02052-6
Beta-amyloid pathology in human brain microvessel extracts from the parietal cortex: relation with cerebral amyloid angiopathy and Alzheimer’s disease
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01967-4
Recurrent KBTBD4 small in-frame insertions and absence of DROSHA deletion or DICER1 mutation differentiate pineal parenchymal tumor of intermediate differentiation (PPTID) from pineoblastoma
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01990-5
Oral and intravenous transmission of α-synuclein fibrils to mice
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02037-5
Neurotoxicity of pesticides
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02033-9
Mitochondrial defect in muscle precedes neuromuscular junction degeneration and motor neuron death in CHCHD10S59L/+ mouse
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01988-z
Restoring brain cholesterol turnover improves autophagy and has therapeutic potential in mouse models of spinocerebellar ataxia
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02019-7
An update on the central nervous system manifestations of neurofibromatosis type 1
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02002-2
The neuropathology of fatal encephalomyelitis in human Borna virus infection
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02047-3
Splicing repression is a major function of TDP-43 in motor neurons
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02042-8
Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02082-0
Dipeptide repeat (DPR) pathology in the skeletal muscle of ALS patients with C9ORF72 repeat expansion
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02050-8
YAP1-fusions in pediatric NF2-wildtype meningioma
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02095-9
The TMEM106B FTLD-protective variant, rs1990621, is also associated with increased neuronal proportion
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02066-0
Loss of DPP6 in neurodegenerative dementia: a genetic player in the dysfunction of neuronal excitability
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01976-3
Caspase-4 mediates cytoplasmic accumulation of TDP-43 in the primate brains
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01979-0
Naturally occurring antibodies isolated from PD patients inhibit synuclein seeding in vitro and recognize Lewy pathology
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01974-5
Vitamin D increases glucocorticoid efficacy via inhibition of mTORC1 in experimental models of multiple sclerosis
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02018-8
Severe bornavirus-encephalitis presenting as Guillain–Barré-syndrome
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02005-z
An update on the central nervous system manifestations of Li–Fraumeni syndrome
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02055-3
Molecular subgrouping of primary pineal parenchymal tumors reveals distinct subtypes correlated with clinical parameters and genetic alterations
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02101-0
Neuroinflammation, the thread connecting neurological disease
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02009-9
Alpha-synuclein: prion or prion-like?
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02057-1
Production of poly(GA) in C9ORF72 patient motor neurons derived from induced pluripotent stem cells
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02083-z
Evidence of corticofugal tau spreading in patients with frontotemporal dementia
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02075-z
Translational control in brain pathologies: biological significance and therapeutic opportunities
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01971-8
A single-center study of the clinicopathologic correlates of gliomas with a MYB or MYBL1 alteration
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02081-1
Structural and functional conservation of non-lumenized lymphatic endothelial cells in the mammalian leptomeninges
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02091-z
Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02109-6
Detection of AD-specific four repeat tau with deamidated asparagine residue 279-specific fraction purified from 4R tau polyclonal antibody
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02012-0
Desmoplastic/nodular medulloblastomas (DNMB) and medulloblastomas with extensive nodularity (MBEN) disclose similar epigenetic signatures but different transcriptional profiles
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-01981-6
Desmoplastic myxoid tumor, SMARCB1-mutant: clinical, histopathological and molecular characterization of a pineal region tumor encountered in adolescents and adults
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02094-w
Human subiculo-fornico-mamillary system in Alzheimer’s disease: Tau seeding by the pillar of the fornix
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02108-7
Subcortical TDP-43 pathology patterns validate cortical FTLD-TDP subtypes and demonstrate unique aspects of C9orf72 mutation cases
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02070-4
Lewy-related pathology exhibits two anatomically and genetically distinct progression patterns: a population-based study of Finns aged 85+
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02071-3
Overlapping genetic architecture between Parkinson disease and melanoma
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02110-z
Posterior fossa pilocytic astrocytomas with oligodendroglial features show frequent FGFR1 activation via fusion or mutation
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02097-7
NF1 patient missense variants predict a role for ATM in modifying neurofibroma initiation
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02086-w
Tectal glioma harbors high rates of KRAS G12R and concomitant KRAS and BRAF alterations
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02112-x
Neuroglial stem cell-derived inflammatory pseudotumor (n-SCIPT): clinicopathologic characterization of a novel lesion of the lumbosacral spinal cord and nerve roots following intrathecal allogeneic stem cell intervention
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02089-7
Neurotoxicology: an update on epidemiology, mechanisms, and pathology
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02051-7
Correction to: 4-Repeat tau seeds and templating subtypes as brain and CSF biomarkers of frontotemporal lobar degeneration
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02092-y
Molecular progression of SHH-activated medulloblastomas
来源期刊:Acta NeuropathologicaDOI:10.1007/s00401-019-02022-y

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